The subsiding of the COVID-19 pandemic and the various effects it could have on the health of infected patients in the long
term have prompted researchers to conduct studies in this field. One of the most notable issues is the finding of a relation
ship between SARS-CoV-2 infection and the development of cancer in human organs.
Author Name: Amir Hossein Tabatabaie |
|
Journal of Endocrinology & Research Studies
Myelodysplastic syndrome (MDS) is defined by inadequate hematopoiesis and a risk of progression to acute myeloid
leukemia (AML). This case report describes a 59-year-old individual with MDS who has a complicated karyotype that in
cludes chromosomal 1q triplication (q21q32) and grade 2 myelofibrosis (MF-2). The patient had severe cytopenias, recur
ring infections, and inflammatory lesions suggestive with pyoderma gangrenosum
Author Name: Bibhas Kar |
|
Journal of Hematology and Blood Disorders
Pulmonary s is a multifactorial chronic progressive disease, caused by numerous biotic and abiotic agents, including
SARS-CoV-2 infection. Damage of the alveolar epithelial barrier (formed by alveolar epithelial cells type I, consisting of
tight and adherens junctions) has been described as a key mechanism of early s and therefore could be crucial for
post-acute sequelae of COVID-19 as well. Integrity of adherens junctions is regulated by the intracellular plaque protein
p120 catenin and is crucial for epithelial barrier regulation in epithelial-mesenchymal-transition-driven diseases like
nesis and cancer progression
Author Name: Julián Wiegner |
|
Journal of Cell Biology and Histology
Staphylococcus aureus and Staphylococcus epidermidis are major agents of nosocomial infections and exhibit high levels of antimicrobial resistance. e CRISPR/Cas system functions as a bacterial defense mechanism against mobile genetic elements, such as phages and plasmids, and may be the acquisition of resistance genes. s study aimed to investigate the presence, diversity, and functional implications of the CRISPR/Cas system in complete genomes of S. aureus and S. epidermidis, including its relationship with integrated bacteriophages, anti-CRISPR genes, and antimicrobial resistance genes. A total of 30 complete genomes from each species were analyzed, all retrieved from the NCBI database
Author Name: Tereza Cristina Leal-Balbino |
|
Journal of Microbiology and Modern Techniques
Loss or Strategy? The Crispr/Cas System and Its Genomic Trade-offs in Staphylococcus aureus and S. epidermidis
The subsiding of the COVID-19 pandemic and the various effects it could have on the health of infected patients in the long
term have prompted researchers to conduct studies in this field. One of the most notable issues is the finding of a relation
ship between SARS-CoV-2 infection and the development of cancer in human organs.
Author Name: Amir Hossein Tabatabaie |
|
Loss or Strategy? The Crispr/Cas System and Its Genomic Trade-offs in Staphylococcus aureus and S. epidermidis
Myelodysplastic syndrome (MDS) is defined by inadequate hematopoiesis and a risk of progression to acute myeloid
leukemia (AML). This case report describes a 59-year-old individual with MDS who has a complicated karyotype that in
cludes chromosomal 1q triplication (q21q32) and grade 2 myelofibrosis (MF-2). The patient had severe cytopenias, recur
ring infections, and inflammatory lesions suggestive with pyoderma gangrenosum
Author Name: Bibhas Kar |
|
Loss or Strategy? The Crispr/Cas System and Its Genomic Trade-offs in Staphylococcus aureus and S. epidermidis
Pulmonary s is a multifactorial chronic progressive disease, caused by numerous biotic and abiotic agents, including
SARS-CoV-2 infection. Damage of the alveolar epithelial barrier (formed by alveolar epithelial cells type I, consisting of
tight and adherens junctions) has been described as a key mechanism of early s and therefore could be crucial for
post-acute sequelae of COVID-19 as well. Integrity of adherens junctions is regulated by the intracellular plaque protein
p120 catenin and is crucial for epithelial barrier regulation in epithelial-mesenchymal-transition-driven diseases like
nesis and cancer progression
Author Name: Julián Wiegner |
|
Loss or Strategy? The Crispr/Cas System and Its Genomic Trade-offs in Staphylococcus aureus and S. epidermidis
Staphylococcus aureus and Staphylococcus epidermidis are major agents of nosocomial infections and exhibit high levels of antimicrobial resistance. e CRISPR/Cas system functions as a bacterial defense mechanism against mobile genetic elements, such as phages and plasmids, and may be the acquisition of resistance genes. s study aimed to investigate the presence, diversity, and functional implications of the CRISPR/Cas system in complete genomes of S. aureus and S. epidermidis, including its relationship with integrated bacteriophages, anti-CRISPR genes, and antimicrobial resistance genes. A total of 30 complete genomes from each species were analyzed, all retrieved from the NCBI database
Author Name: Tereza Cristina Leal-Balbino |
|